Crixivan and Biaxin drug interactions - a phase IV clinical study of FDA data

Summary:

Drug interactions are reported among people who take Crixivan (indinavir sulfate) and Biaxin (clarithromycin). Common drug interactions include blood urea increased among females and diabetic ketoacidosis among males.

The phase IV clinical study analyzes what interactions people have when they take Crixivan and Biaxin. It is created by eHealthMe based on reports of 56 people who take the same drugs from the FDA, and is updated regularly.

What is Crixivan?

Crixivan has active ingredients of indinavir sulfate. eHealthMe is studying from 6,312 Crixivan users. Check the latest studies of Crixivan.

What is Biaxin?

Biaxin has active ingredients of clarithromycin. It is often used in sinusitis. eHealthMe is studying from 14,248 Biaxin users. Check the latest studies of Biaxin.

On Jul, 04, 2026

56 people who take Crixivan and Biaxin together, and have interactions are studied.

What are the common drug interactions of Crixivan and Biaxin, by gender? *:

female:

Blood urea increased
Cholecystitis (infection of gallbladder)
Diarrhoea
Gastric dilatation (a twisted stomach)
Haematuria present (presence of blood in urine due to trauma)
Lymphoma (cancer that begins in immune system cells)
Multiple myeloma (cancer of the plasma cells)
Oedema (fluid collection in tissue)
Proteinuria present
Pyrexia (fever)

male:

Diabetic ketoacidosis (diabetic ketoacidosis (dka) is high concentrations of ketone bodies)
Vomiting
Anaemia (lack of blood)
Blood amylase increased
Blood glucose increased
Fat disorder (inability to produce fat utilize enzyme)
Hypercholesterolaemia (high levels of cholesterol in the blood)
Hypertriglyceridaemia (excess of triglycerides in the blood)
Jaundice (a yellowish pigmentation of the skin, the conjunctival membranes)
Lactic acidosis (low ph in body tissues)

What are the common drug interactions of Crixivan and Biaxin, by age (0-1 to 60+)? *:

0-1:

n/a

2-9:

n/a

10-19:

Pancreatitis necrotising (inflammation of the pancreas cause death of tissue in pancreas)

20-29:

n/a

30-39:

Gastrointestinal disorder (functional problems of gastrointestinal tract)
Oedema peripheral (superficial swelling)
Pulmonary hypertension (increase in blood pressure in the lung artery)
Right ventricular failure (right half of the heart fails to work)
Abdominal distension
Blood glucose increased
Blood triglycerides increased
Cholecystitis (infection of gallbladder)
Depression
Gastric dilatation (a twisted stomach)

40-49:

Anaemia (lack of blood)
Acute abdomen
Coma (state of unconsciousness lasting more than six hours)
Depressed level of consciousness
Hepatitis (inflammation of the liver)
Hepatocellular damage (liver damage)
Jaundice (a yellowish pigmentation of the skin, the conjunctival membranes)
Renal failure acute (rapid kidney dysfunction)
Thrombocytopenia (decrease of platelets in blood)
Blood creatinine increased

50-59:

Renal failure (kidney dysfunction)
Blood creatinine increased
Blood urea increased
Diarrhoea
Gastrointestinal disorder (functional problems of gastrointestinal tract)
Haematuria present (presence of blood in urine due to trauma)
Multiple myeloma (cancer of the plasma cells)
Proteinuria present
Vomiting
Blood glucose increased

60+:

Abdominal pain
Blood amylase increased
Fat disorder (inability to produce fat utilize enzyme)
Hyperglycaemia (high blood sugar)
Taste disturbance
Diabetes mellitus (diabetes, caused by a deficiency of the pancreatic hormone insulin)
Diabetic ketoacidosis (diabetic ketoacidosis (dka) is high concentrations of ketone bodies)
Lipase increased
Nausea (feeling of having an urge to vomit)
Pancreatitis (inflammation of pancreas)

* Approximation only. Some reports may have incomplete information.

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